Gene: SOD1 ×
Source: ALL
Gene Score gda Association Type Type Original DB Sentence supporting the association PMID PMID Year
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Biomarker disease CTD_human Lycium barbarum polysaccharides reduce intestinal ischemia/reperfusion injuries in rats. 23743330 2013
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Therapeutic disease CTD_human Lycium barbarum polysaccharides reduce intestinal ischemia/reperfusion injuries in rats. 23743330 2013
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Biomarker disease CTD_human NO-mediated S-nitrosylation of PDI may be involved in the formation of the SOD1-linked ubiquitinated-protein aggregates in cerebral ischemia/reperfusion injury. 23061969 2012
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Therapeutic disease CTD_human NO-mediated S-nitrosylation of PDI may be involved in the formation of the SOD1-linked ubiquitinated-protein aggregates in cerebral ischemia/reperfusion injury. 23061969 2012
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Biomarker disease LHGDN Early vulnerability to ischemia/reperfusion injury in motor terminals innervating fast muscles of SOD1-G93A mice. 17292357 2007
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Biomarker disease CTD_human These results suggest that overexpression of SOD1 may affect the PRAS pathway after tFCI by reducing the direct oxidative reaction to pPRAS after reperfusion injury. 16397181 2006
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Therapeutic disease CTD_human These results suggest that overexpression of SOD1 may affect the PRAS pathway after tFCI by reducing the direct oxidative reaction to pPRAS after reperfusion injury. 16397181 2006
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Therapeutic disease CTD_human We hypothesized that overexpression of SOD1 in donor hearts would suppress ischemia-reperfusion injury and thereby reduce GCAD. 15364863 2004
Entrez Id: 6647
Gene Symbol: SOD1
SOD1 		0.310 Biomarker disease CTD_human We hypothesized that overexpression of SOD1 in donor hearts would suppress ischemia-reperfusion injury and thereby reduce GCAD. 15364863 2004