Source: ALL
Disease Score gda Association Type Type Original DB Sentence supporting the association PMID PMID Year
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 Biomarker group BEFREE Although this result is currently a matter of controversy, further studies must be independently repeated to be conclusive; -- another RFLP was found in c-mos proto-oncogene, which is detected only in patients with breast cancers or other types of tumors. 2905174 1988
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 Biomarker group BEFREE There was up to 5-fold enrichment of the targeted MSV in the bone marrow of mice bearing early or late stage MDA-MB-231 tumors and of mice with late stage, but not early stage, MCF-7 tumors. 24818768 2014
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 AlteredExpression group BEFREE We found c-mos overexpression in 27% of the tumors. 11212247 2001
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 Biomarker group BEFREE How this process occurs and more importantly, why, has recently become the focus of several research groups, prompting this review in which we discuss two related phenomena that accompany the reversible polyploidy of tumor cells: the induction of meiosis genes such as MOS and the decrease in genomic instability observed during the reversion from polyploidy to para-diploidy. 20676137 2010
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 Biomarker group BEFREE Female transgenic mice lacking a functional c-mos proto-oncogene develop ovarian teratomas, indicating that c-mos may behave as a tumour-suppressor gene for this type of tumour. 9635841 1998
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 Biomarker group BEFREE In the present report, we examine, in a series of non-small cell lung carcinomas (NSCLCs), the status of K-ras and c-mos oncoproteins in correlation with the tumor neo-angiogenesis state and the major angiogenic factor, vascular endothelial growth factor (VEGF). 11778648 2001
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 GeneticVariation group BEFREE Thus, trisomy 8 associated with human hematologic neoplasia is generally not related to gross rearrangements of the c-mos or c-myc genes. 4079453 1985
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 GeneticVariation group BEFREE We carried out direct sequence analysis of human c-mos and human ENRB in a series of sporadic MTC and phaeochromocytomas to determine if somatic mutations in these two genes could account for some of the sporadic MEN 2-related tumours in which no RET mutations are detected.No somatic mutations were found. 8695346 1996
CUI: C0027651
Disease: Neoplasms
Neoplasms
0.090 Biomarker group BEFREE Rhim et al. were first to show that superinfection of Ad12-SV40-infected immortalized human epidermal cells with an RNA tumor virus containing a ras oncogene, such as Ki-MSV, or their treatment with chemical carcinogens, leads to the ability of cells to both grow in anchorage-independent fashion and to form tumors in athymic nude mice. 8330279 1993