<b>Background:</b> Transforming growth factor-beta-induced (TGFBI) is an exocrine protein, which has been found to be able to promote the development of nasopharyngeal carcinoma, glioma, pancreatic cancer, and other tumors.
<b>Conclusion:</b> Sev impeded cell migration and invasion through regulating miR-146b-5p and MMP16 in glioma, indicating a novel theories foundation for the application of anesthetics like Sev in glioma.
<b>Conclusion:</b> SUV quantification of <sup>18</sup>F-FLT uptake in glioma had an RC in the range of 18%-24% when imaging began 1 h after <sup>18</sup>F-FLT administration.
<b>Conclusion:</b> The synergistic effects, such as a higher inhibition rate, and lower expressions of MRP1 and VEGF, of combined curcumin and LFLIU against glioma was much better than that of a single treatment.
<b>Conclusion:</b> The synergistic effects, such as a higher inhibition rate, and lower expressions of MRP1 and VEGF, of combined curcumin and LFLIU against glioma was much better than that of a single treatment.
<b>Conclusion:</b> The synergistic effects, such as a higher inhibition rate, and lower expressions of MRP1 and VEGF, of combined curcumin and LFLIU against glioma was much better than that of a single treatment.
<b>Conclusion:</b> These results demonstrated that LAMC1 may play an important role in glioma progression and may be used in the diagnosis, prognosis, and targeted therapy of glioma patients.
<b>Conclusions:</b> This study suggests that HOXC10 induces glioma angiogenesis by transcriptionally upregulating <i>VEGFA</i> expression, and may represent a potential target for antiangiogenic therapy in gliomas.
<b>Methods:</b> Patients with a diagnosis of World Health Organization (WHO) Grade I-IVglioma treated with definitive radiation therapy (RT) between the years of 2004-13 were identified.
<b>Methods:</b> We compared 50 <sup>18</sup>F-FDG PET/CT studies collected prospectively from 14 patients with supratentorial glioma (5 men and 9 women; age range, 35-77 y) with 10 single scans from healthy controls (age range, 43-75 y).
<b>Patients and methods:</b> qRT-PCR was used to determine the expression of miR-145 in glioma patients and GSCs, and GSCs were transfected with miR-145 overexpressed vectors.
<b>Results:</b> Five pseudogenes (ANXA2P2, EEF1A1P9, FER1L4, HILS1, and RAET1K) correlating with glioma survival were selected and used to establish a risk signature.
<i>FoxD2-AS1</i> is a prognostic factor in glioma and promotes temozolomide resistance in a O<sup>6</sup>-methylguanine-DNA methyltransferase-dependent manner.