Gene Score gda Association Type Type Original DB Sentence supporting the association PMID PMID Year
Entrez Id: 4846
Gene Symbol: NOS3
NOS3
0.500 Biomarker phenotype CTD_human These data highlight the potential utility of eNOS gene therapy in the treatment of hypertension and insulin resistance. 19008412 2009
Entrez Id: 4846
Gene Symbol: NOS3
NOS3
0.500 Therapeutic phenotype CTD_human These data highlight the potential utility of eNOS gene therapy in the treatment of hypertension and insulin resistance. 19008412 2009
Entrez Id: 4846
Gene Symbol: NOS3
NOS3
0.500 Biomarker phenotype CTD_human In conclusion, we described a significant association between eNOS gene polymorphisms and type 2 diabetes, suggesting a new genetic susceptibility factor for hyperinsulinemia, insulin resistance, and type 2 diabetes. 12716763 2003
Entrez Id: 4846
Gene Symbol: NOS3
NOS3
0.500 Therapeutic phenotype CTD_human Clustering of cardiovascular risk factors mimicking the human metabolic syndrome X in eNOS null mice. 12947532 2003
Entrez Id: 4846
Gene Symbol: NOS3
NOS3
0.500 Biomarker phenotype CTD_human Clustering of cardiovascular risk factors mimicking the human metabolic syndrome X in eNOS null mice. 12947532 2003
Entrez Id: 4846
Gene Symbol: NOS3
NOS3
0.500 Therapeutic phenotype CTD_human In conclusion, we described a significant association between eNOS gene polymorphisms and type 2 diabetes, suggesting a new genetic susceptibility factor for hyperinsulinemia, insulin resistance, and type 2 diabetes. 12716763 2003
Entrez Id: 4846
Gene Symbol: NOS3
NOS3
0.500 Biomarker phenotype CTD_human Insulin resistance in eNOS(-/-) mice was related specifically to impaired NO synthesis, because in equally hypertensive 1-kidney/1-clip mice (a model of renovascular hypertension), insulin-stimulated glucose uptake was normal. 11457755 2001
Entrez Id: 4846
Gene Symbol: NOS3
NOS3
0.500 Therapeutic phenotype CTD_human Insulin resistance in eNOS(-/-) mice was related specifically to impaired NO synthesis, because in equally hypertensive 1-kidney/1-clip mice (a model of renovascular hypertension), insulin-stimulated glucose uptake was normal. 11457755 2001