In conclusion, our pooled systematic study results indicated that individuals with the A allele h</span>ad a higher risk of developing VTEthan those with the C allele of the rs13146272 variant, but the risk was inconsistent among different ethnicities.
Finally, our study demonstrated the important role of rs2289252, rs2036914, rs2066865, and rs13146272 polymorphisms in the development of VTE in the white race.
However, using this large cohort of subjects, we were able to replicate the mild effects of 2 nonsynonymous SNPs, rs1613662 in GP6 and rs13146272 in CYP4V2, recently suspected to be associated with VTE.