|
Hypertriglyceridemia
|
0.400 |
GeneticVariation
|
phenotype |
BEFREE |
Low total testosterone levels were correlated with lower high-density lipoprotein cholesterol and higher triglycerides, high-sensitivity C-reactive protein, high-sensitivity troponin T, N-terminal-pro B-type natriuretic peptide and glucose levels (all p < 0.01).
|
31154826 |
2019 |
|
Hypertriglyceridemia
|
0.400 |
Biomarker
|
phenotype |
BEFREE |
In the multivariable analysis, plasma CRP concentration was associated with increased prevalence of hypertriglyceridaemia (OR 1.157, 95% CI 1.040 to 1.287, p=0.007), diabetes (OR 1.204, 95% CI 1.058 to 1.371, p=0.005) and metabolic syndrome (OR 1.228, 95% CI 1.112 to 1.357, p<0.001) after adjustment for socioeconomic and lifestyle characteristics.
|
31473619 |
2019 |
|
Hypertriglyceridemia
|
0.400 |
GeneticVariation
|
phenotype |
BEFREE |
Older age, cigarette smoking, increased BMI, kidney dysfunction, and elevated triglycerides are associated with greater risk of mitral annular calcification, but conflicting evidence exists for sex and C-reactive protein.
|
30694830 |
2019 |
|
Hypertriglyceridemia
|
0.400 |
Biomarker
|
phenotype |
BEFREE |
With EPA and DHA intake varying between 0.03 to 5 g per day, biomarkers, such as triglycerides, high-density lipoprotein and platelet aggregation, tended to ameliorate when daily intake exceeded 1 g per day, while the most common inflammatory marker, C-reactive protein, was not affected.
|
29420061 |
2019 |
|
Hypertriglyceridemia
|
0.400 |
Biomarker
|
phenotype |
BEFREE |
For all subclasses, a low level of galactosylation and sialylation and a high degree of core fucosylation associated with poor metabolic health, i.e. increased inflammation as assessed by C-reactive protein, low serum high-density lipoprotein cholesterol and high triglycerides, which are all known to indicate increased risk of cardiovascular disease.
|
28951559 |
2017 |
|
Hypertriglyceridemia
|
0.400 |
Biomarker
|
phenotype |
BEFREE |
Of the metabolic syndrome components, elevated waist circumference, low high-density lipoprotein-cholesterol and high triglycerides were significantly related to CRP in a graded (dose-response) manner.
|
28537577 |
2017 |
|
Hypertriglyceridemia
|
0.400 |
GeneticVariation
|
phenotype |
BEFREE |
Once demographics, medication use and baseline adiposity, and fitness were accounted for, ILI did not modify the baseline association of GCKR-Leu446Pro with elevated triglycerides (β±SE=0.067±0.013, P=1.5×10(-7) and β±SE=0.052±0.015, P=5×10(-4)) or with elevated CRP (β±SE=0.136±0.034, P=5.1×10(-5)and β±SE=0.903±0.038, P=0.015) in the overall sample and Non-Hispanic Whites, respectively.
|
26578543 |
2016 |
|
Hypertriglyceridemia
|
0.400 |
Biomarker
|
phenotype |
BEFREE |
AD patients had a more atherogenic lipid profile characterized by an increase in the prevalence of small dense LDL (p = 0.003), increased triglycerides (p = 0.002), reduced HDLC (p<0.001) an elevated highly sensitive C-reactive protein (p = 0.01), compared with controls.
|
24466047 |
2014 |
|
Hypertriglyceridemia
|
0.400 |
GeneticVariation
|
phenotype |
BEFREE |
We tested whether low high-density lipoprotein cholesterol (HDL-C) and/or high triglycerides are associated to abnormal HDL subclasses distribution and composition, and their relationships with fasting insulin and C-reactive protein (CRP).
|
18420210 |
2008 |
|
Hypertriglyceridemia
|
0.400 |
Biomarker
|
phenotype |
BEFREE |
There were no clear indications of synergistic interaction effects involving the PAI-1 4G/5G polymorphism and the environmental exposures considered (cigarette smoking, physical inactivity, overweight, diabetes mellitus, hypercholesterolaemia, hypertension, high C-reactive protein and hypertriglyceridaemia).
|
12783120 |
2003 |
|
Hypertriglyceridemia
|
0.400 |
Therapeutic
|
phenotype |
CTD_human |
Severe hypertriglyceridemia with insulin resistance is associated with systemic inflammation: reversal with bezafibrate therapy in a randomized controlled trial.
|
11893366 |
2002 |