|
Respiratory Syncytial Virus Infections
|
0.100 |
Biomarker
|
group |
BEFREE |
In conclusion, we confirm that RSV infection leads to the increased expression of 12/15 LOX and the related chemokines CCL5 and CCL3 in BAL fluid and lung tissue cells suggesting that the 12/15 LOX pathway could serve as a candidate target for prevention and treatment of RSV infection.
|
28579398 |
2017 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
AlteredExpression
|
group |
BEFREE |
Full-term children with HMPV and RSV infection had increased levels of nasal airway IFNγ, CCL5, and IL-10 along with an elevation in Th1 (IFNγ)/Th2 (IL-4) ratios, which is expected during antiviral responses.
|
26086642 |
2015 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
Biomarker
|
group |
BEFREE |
The amount of MMP-10 released from HNECs was also increased in a time-dependent manner after RSV infection as was that of chemokine RANTES.
|
24009192 |
2013 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
AlteredExpression
|
group |
BEFREE |
Independent determinants significantly associated with increased risk for physician-diagnosed asthma by the seventh birthday included maternal asthma (odds ratio [OR], 5.2; 95% CI, 1.7-15.9; P = .004), exposure to high levels of dog allergen (OR, 3.2; 95% CI, 1.3-7.7; P = .012), aeroallergen sensitivity at age 3 years (OR, 10.7; 95% CI, 2.1-55.0; P = .005), recurrent wheezing during the first 3 years of life (OR, 7.3; 95% CI, 1.2-43.3; P = .028), and CCL5 expression in nasal epithelia during acute RSV infection (OR, 3.8; 95% CI, 1.2-2.4; P < .001).
|
22444510 |
2012 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
Biomarker
|
group |
BEFREE |
RSV infection of WD-NE resulted in significant accumulations of interleukin (IL)-6, IL-8, and RANTES when compared with findings in control samples.
|
15867636 |
2005 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
Biomarker
|
group |
BEFREE |
Inhibition of respiratory syncytial virus infection with the CC chemokine RANTES (CCL5).
|
15122808 |
2004 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
Biomarker
|
group |
BEFREE |
Our results show that RSV-infected airway epithelial cells and alveolar macrophages display differential profiles of chemokine production: airway epithelial cells produce CCL2/monocyte chemoattractant protein-1, CCL5/RANTES, CXCL10/gamma interferon inducible protein-10, and kerotinocyte cytokine (KC); and alveolar macrophages up-regulate CCL5 and macrophage inflammatory protein (MIP)-2 after RSV infection.
|
15073679 |
2004 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
Biomarker
|
group |
BEFREE |
Patients hospitalized because of RSV infection had increased numbers of CD16(+) and CD56(bright) cells and had RSV-specific increases in Th1 (interleukin [IL]-2 and interferon-gamma) and Th2 (IL-4 and IL-6) cytokines and CC chemokines (macrophage inflammatory protein [MIP]-1alpha, MIP-1beta, and RANTES [regulated on activation, normally T cell expressed and secreted]) mRNA expression.
|
11992272 |
2002 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
AlteredExpression
|
group |
BEFREE |
The signaling pathway regulating RANTES gene expression after RSV infection has not been determined.
|
12114198 |
2002 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
Biomarker
|
group |
BEFREE |
We conclude that chemokines IL-8, RANTES, MIP-1alpha, and MCP-1 are all increased in nasal secretions in human RSV infection at the time of virus shedding and symptomatic illness and that the epithelium lining the nasal turbinate contributes to the increase in RANTES.
|
10998316 |
2000 |
|
Respiratory Syncytial Virus Infections
|
0.100 |
AlteredExpression
|
group |
BEFREE |
RANTES (regulated upon activation, normal T cell-expressed and -secreted), which was present in negligible concentrations in uninfected cultures, was strongly induced by RSV infection, in a dose- and time-dependent manner.
|
9041319 |
1997 |